Investigators are sounding a new warning about NSAIDs and acute kidney injury in children.
Severe acute kidney injuries have been rising rapidly in the United States over the past 10 years.
The incidence of AKI requiring dialysis is now higher than the incidence of ESRD requiring dialysis or transplant.
Condition significantly increases the risk of cardiovascular events and death.
AKI develops in 7% of patients undergoing PCI.
More than 60% of patients had died within four years of entry into a clinical trial of different RRT intensities for acute kidney injury.
Patients found to have a 67% increased risk of coronary events or death compared with patients who did not suffer acute kidney injury.
Acute kidney injury seen in 7% of patients undergoing percutaneous coronary intervention.
Lithium promotes kidney repair in mouse models.
Patients hospitalized with AKI experienced more adverse renocardiovascular events than patients hospitalized with MI.
Absence of a link found in a pooled analysis of two trials with a total of about 8,600 participants.
Hospitalizations for acute kidney injury rose along with increased use of ACE inhibitors and angiotensin receptor antagonists.
ACE inhibitor plus ARB failed to slow disease progression but increased acute kidney injury risk.
The antibiotic inhibits an enzyme that metabolizes calcium-channel blockers, raising levels of these drugs.
Patients whose statins were held in the 24 hours before cardiac surgery had higher levels of kidney injury biomarkers.
After adjusting for health insurance and income, African Americans no longer had a significantly elevated risk than Caucasians.
Infants aged one month or younger who experience acute kidney injury during hospitalization have a 31% death rate.
A prospective cohort study has shown plasma NGAL to be an accurate biomarker for predicting acute kidney injury.